Open Access Articles- Top Results for ZBTB17


SymbolsZBTB17 ; MIZ-1; ZNF151; ZNF60; pHZ-67
External IDsOMIM604084 MGI107410 HomoloGene2575 GeneCards: ZBTB17 Gene
RNA expression pattern
File:PBB GE ZBTB17 203602 s at tn.png
File:PBB GE ZBTB17 203601 s at tn.png
More reference expression data
RefSeq (mRNA)NM_001242884NM_009541
RefSeq (protein)NP_001229813NP_033567
Location (UCSC)Chr 1:
16.27 – 16.3 Mb
Chr 4:
141.44 – 141.47 Mb
PubMed search[1][2]

Zinc finger and BTB domain-containing protein 17 is a protein that in humans is encoded by the ZBTB17 gene.[1]


ZBTB17 has been shown to interact with TOPBP1,[2] Host cell factor C1[3] and Myc.[4][5]


  1. ^ "Entrez Gene: ZBTB17 zinc finger and BTB domain containing 17". 
  2. ^ Herold, Steffi; Wanzel Michael; Beuger Vincent; Frohme Carsten; Beul Dorothee; Hillukkala Tomi; Syvaoja Juhani; Saluz Hans-Peter; Haenel Frank; Eilers Martin (September 2002). "Negative regulation of the mammalian UV response by Myc through association with Miz-1". Mol. Cell (United States) 10 (3): 509–21. ISSN 1097-2765. PMID 12408820. doi:10.1016/S1097-2765(02)00633-0. 
  3. ^ Piluso, David; Bilan Patricia; Capone John P (November 2002). "Host cell factor-1 interacts with and antagonizes transactivation by the cell cycle regulatory factor Miz-1". J. Biol. Chem. (United States) 277 (48): 46799–808. ISSN 0021-9258. PMID 12244100. doi:10.1074/jbc.M206226200. 
  4. ^ Staller, P; Peukert K, Kiermaier A, Seoane J, Lukas J, Karsunky H, Möröy T, Bartek J, Massagué J, Hänel F, Eilers M (April 2001). "Repression of p15INK4b expression by Myc through association with Miz-1". Nat. Cell Biol. (England) 3 (4): 392–9. ISSN 1465-7392. PMID 11283613. doi:10.1038/35070076. 
  5. ^ Peukert, K; Staller P; Schneider A; Carmichael G; Hänel F; Eilers M (September 1997). "An alternative pathway for gene regulation by Myc". EMBO J. (ENGLAND) 16 (18): 5672–86. ISSN 0261-4189. PMC 1170199. PMID 9312026. doi:10.1093/emboj/16.18.5672. 

Further reading


External links

This article incorporates text from the United States National Library of Medicine, which is in the public domain.

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