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Open Access Articles- Top Results for ZNF148

ZNF148

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Identifiers
SymbolsZNF148 ; BERF-1; BFCOL1; HT-BETA; ZBP-89; ZFP148; pHZ-52
External IDsOMIM601897 MGI1332234 HomoloGene8003 GeneCards: ZNF148 Gene
Orthologs
SpeciesHumanMouse
Entrez770722661
EnsemblENSG00000163848ENSMUSG00000022811
UniProtQ9UQR1Q61624
RefSeq (mRNA)NM_021964NM_011749
RefSeq (protein)NP_068799NP_035879
Location (UCSC)Chr 3:
124.94 – 125.09 Mb
Chr 16:
33.38 – 33.5 Mb
PubMed search[1][2]

Zinc finger protein 148 is a protein that in humans is encoded by the ZNF148 gene.[1][2][3]


Interactions

ZNF148 has been shown to interact with PTRF[4] and P53.[5]

See also

References

  1. ^ Tommerup N, Vissing H (November 1995). "Isolation and fine mapping of 16 novel human zinc finger-encoding cDNAs identify putative candidate genes for developmental and malignant disorders". Genomics 27 (2): 259–64. PMID 7557990. doi:10.1006/geno.1995.1040. 
  2. ^ Antona V, Cammarata G, De Gregorio L, Dragani TA, Giallongo A, Feo S (March 1999). "The gene encoding the transcriptional repressor BERF-1 maps to a region of conserved synteny on mouse chromosome 16 and human chromosome 3 and a related pseudogene maps to mouse chromosome 8". Cytogenet Cell Genet 83 (1–2): 90–2. PMID 9925940. doi:10.1159/000015138. 
  3. ^ "Entrez Gene: ZNF148 zinc finger protein 148". 
  4. ^ Hasegawa, T; Takeuchi A; Miyaishi O; Xiao H; Mao J; Isobe K (April 2000). "PTRF (polymerase I and transcript-release factor) is tissue-specific and interacts with the BFCOL1 (binding factor of a type-I collagen promoter) zinc-finger transcription factor which binds to the two mouse type-I collagen gene promoters". Biochem. J. (ENGLAND). 347 Pt 1 (Pt 1): 55–9. ISSN 0264-6021. PMC 1220930. PMID 10727401. doi:10.1042/0264-6021:3470055. 
  5. ^ Bai, L; Merchant J L (July 2001). "ZBP-89 Promotes Growth Arrest through Stabilization of p53". Mol. Cell. Biol. (United States) 21 (14): 4670–83. ISSN 0270-7306. PMC 87140. PMID 11416144. doi:10.1128/MCB.21.14.4670-4683.2001. 

Further reading

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External links

This article incorporates text from the United States National Library of Medicine, which is in the public domain.



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